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International Immunology 2008 20(10):NP; doi:10.1093/intimm/dxn107
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© The Japanese Society for Immunology. 2008. All rights reserved. For permissions, please e-mail: journals.permissions@oxfordjournals.org

IN THIS ISSUE

The first 10% of the full text of this article appears below.


   Complement peptide inhibits mast cell responses
 
Complement components can activate certain mast cell functions. Here, Erdei and colleagues (p. 1239) show that C3a9 — a peptide derived from C3a — binds mast cell Fc{varepsilon}RI–IgE complexes, affecting receptor internalisation and the phosphorylation of various kinases; it also affects cytokine secretion. This fragment therefore inhibits both the immediate and late-phase responses of mast cells.


   Different TCR–CD3 structures on CD4+ and CD8+ cells
 
Previously, Regueiro and colleagues (p. 1247) showed that antibodies can distinguish the {alpha}. . . [Full Text of this Article]


   DC-STAMP regulates autoimmunity and DC function
 

   A new model for T helper subset differentiation
 

   Fas expression and germinal centre tolerance induction
 

   Selective role of Gads in allergy
 

   Altered immunity in D4-GDI transgenic mice
 

   Glatiramer acetate has little effect in lupus
 

   BCG inhibits Th2 responses
 

   T cell clones mediate both arthritis and pneumonitis
 

   NK cells help mediate skin allograft rejection
 

   Tissue damage may trigger elimination of tissue-specific B cells
 

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